The etiology of this disease can be:
In the presence of heart disease. These include: chronic ischemia of the heart; myocardial infarction; inflammation of the heart muscle; heart defects; cardiosclerosis; In case of poisoning with drugs: glycosides; adrenoblockers; antiarrhythmic drugs; As a result of a decrease in the production of thyroid hormones; Acute myocardial infarction; angina; Neoplasms in the heart; Endocarditis; Amyloidosis, sarcoidosis; Degenerative progressive fibrosis and calcification of Meclizine structures; Infectious process (for example, endocarditis, rheumatism); Due to heart surgery.
Complete AV block can be congenital or acquired.
Congenital AV block is usually associated with impaired conduction in the AV node.
The patient may have no symptoms at rest, or they may be minimal, but due to the fixed heart rate, there may be poor exercise tolerance.
Congenital AV block in patients without structural heart abnormalities is often associated with a particular type of maternal antibody.
The causes of acquired AV blockade are as follows: An overdose of one drug that can slow down AV conduction, or the combined use of several (for example, the simultaneous use of a beta-blocker and verapamil). meclizine pills can occur under the influence of class Ia antiarrhythmics (quinidine, procainamide), class Ic (propafenone, ethacizin, flecainide), class II (beta-blockers), class III (amiodarone, sotalol), class IV (calcium antagonists verapamil and diltiazem) , cardiac glycosides. Other causes: Myocardial infarction: an infarction in the anterior wall may be complicated by distal type AV block (due to damage to the branches of the intraventricular conduction system); Inferior myocardial infarction in almost 10% of cases is combined with proximal complete AV block, which is usually unstable and resolves within hours or days.